Meningitis arteritis responsiva a esteroides

Categorical variables were described as fractions or percentages, and comparisons among them were performed by χ 2 test. Nonparametric data were described using median and range with comparisons between paired and nonpaired samples performed by the Wilcoxon signed-rank test and the Mann-Whitney U –test, respectively. Analyses of correlations between the paired variables were performed by calculating Spearman's rank correlation coefficients. For all comparisons, a P value of <.05 was considered to be significant. All statistical analyses were performed by GraphPad. software d

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The diagnosis of the disorder is suggested by the health history and physical examination. A health care professional frequently notes muscle tenderness and that the motion of the shoulders is limited by pain. The joints are usually not swollen. However, signs such as swelling of the small joints of the hands, wrists, and/or knees can occur. Blood testing for inflammation is generally abnormal, as indicated by a significant elevation in the erythrocyte sedimentation rate (sed rate or ESR) and/or C-reactive protein . There are no specific tests, however, for the condition and X-rays are normal. The diagnosis is based on the characteristic history of persisting muscle and joint pain and stiffness associated with elevated blood tests for inflammation, such as the ESR. It is also not unusual for patients to have slight elevations of liver blood tests .

The bacterial flora of brain abscess depends on the source of the infection. In the case of sinusitis and otitis, it is often polymicrobial, including anaerobes. Hematogenous abscesses are most often caused by staphylococcus and streptococcus species. Bacteremia alone does not cause brain abscess. Some tissue damage, probably a small ischemic lesion, is required to start the process. Bacteria in the blood seed this necrotic nidus and spread around it causing brain necrosis and acute inflammation ( cerebritis ). The same early pathology develops from contiguous spread of infection. The necrotic center cavitates while, at the periphery, a vascular zone of brain tissue with macrophages, mononuclear cells, and reactive astrocytes contains the infection. In 4-5 weeks, collagen (derived from vascular cells) is laid down in this reactive zone forming a thick capsule that walls off the infection.

Meningitis arteritis responsiva a esteroides

meningitis arteritis responsiva a esteroides

The bacterial flora of brain abscess depends on the source of the infection. In the case of sinusitis and otitis, it is often polymicrobial, including anaerobes. Hematogenous abscesses are most often caused by staphylococcus and streptococcus species. Bacteremia alone does not cause brain abscess. Some tissue damage, probably a small ischemic lesion, is required to start the process. Bacteria in the blood seed this necrotic nidus and spread around it causing brain necrosis and acute inflammation ( cerebritis ). The same early pathology develops from contiguous spread of infection. The necrotic center cavitates while, at the periphery, a vascular zone of brain tissue with macrophages, mononuclear cells, and reactive astrocytes contains the infection. In 4-5 weeks, collagen (derived from vascular cells) is laid down in this reactive zone forming a thick capsule that walls off the infection.

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